TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis.
第一作者:
Angela,Castoldi
第一单位:
Disciplina de Nefrologia, Departamento de Medicina, Universidade Federal de São Paulo, São Paulo, Brazil.
作者:
主题词
急性肾损伤(Acute Kidney Injury);动物(Animals);细胞因子类(Cytokines);基因缺失(Gene Deletion);肾(Kidney);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);髓样分化因子88(Myeloid Differentiation Factor 88);中性白细胞浸润(Neutrophil Infiltration);中性白细胞(Neutrophils);脓毒症(Sepsis);信号传导(Signal Transduction);Toll样受体2(Toll-Like Receptor 2);Toll样受体4(Toll-Like Receptor 4)
DOI
10.1371/journal.pone.0037584
PMID
22655058
发布时间
2022-03-18
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