Suppression of TRIF-dependent signaling pathway of toll-like receptors by allyl isothiocyanate in RAW 264.7 macrophages.
第一作者:
Soo-Jung,Kim
第一单位:
Department of Biomedical Laboratory Science, College of Medical Sciences, Soonchunhyang University, Asan-Si, Chungnam 336-745, Korea.
作者:
医学主题词
衔接蛋白质类, 膜泡运输(Adaptor Proteins, Vesicular Transport);动物(Animals);消炎药, 非甾类(Anti-Inflammatory Agents, Non-Steroidal);细胞系(Cell Line);趋化因子CXCL10(Chemokine CXCL10);干扰素调节因子3(Interferon Regulatory Factor-3);异硫氰酸盐类(Isothiocyanates);脂多糖类(Lipopolysaccharides);巨噬细胞(Macrophages);小鼠(Mice);NF-κB(NF-kappa B);聚肌胞苷酸(Poly I-C);信号传导(Signal Transduction);Toll样受体(Toll-Like Receptors);转录激活(Transcriptional Activation)
DOI
10.1016/j.intimp.2012.05.017
PMID
22668719
发布时间
2017-11-16
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