Hepatitis B virus-induced lipid alterations contribute to natural killer T cell-dependent protective immunity.
第一作者:
Sebastian,Zeissig
第一单位:
Division of Gastroenterology, Hepatology, and Endoscopy, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts, USA.
作者:
主题词
适应性免疫(Adaptive Immunity);腺病毒科(Adenoviridae);动物(Animals);抗原, CD1d(Antigens, CD1d);载体蛋白质类(Carrier Proteins);共同培养技术(Coculture Techniques);乙型肝炎表面抗原(Hepatitis B Surface Antigens);乙型肝炎病毒(Hepatitis B virus);乙型肝炎, 慢性(Hepatitis B, Chronic);肝细胞(Hepatocytes);人类(Humans);免疫(Immunity);干扰素γ(Interferon-gamma);脂类代谢(Lipid Metabolism);淋巴细胞活化(Lymphocyte Activation);溶血磷脂素类(Lysophospholipids);溶酶体(Lysosomes);小鼠(Mice);自然杀伤T细胞(Natural Killer T-Cells);磷脂酶A2, 分泌型(Phospholipases A2, Secretory)
DOI
10.1038/nm.2811
PMID
22706385
发布时间
2025-05-29
- 浏览18
Nature medicine
1060-8页
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