Stress hematopoiesis reveals abnormal control of self-renewal, lineage bias, and myeloid differentiation in Mll partial tandem duplication (Mll-PTD) hematopoietic stem/progenitor cells.
第一作者:
Yue,Zhang
第一单位:
Division of Pathology, Cincinnati Children's Hospital Medical Center, Cincinnati, OH 45229, USA.
作者:
医学主题词
动物(Animals);细胞分化(Cell Differentiation);细胞谱系(Cell Lineage);细胞增殖(Cell Proliferation);细胞, 培养的(Cells, Cultured);基因复制(Gene Duplication);血细胞生成(Hematopoiesis);造血干细胞(Hematopoietic Stem Cells);组蛋白赖氨酸N-甲基转移酶(Histone-Lysine N-Methyltransferase);人类(Humans);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);髓系细胞(Myeloid Cells);髓淋巴细胞性白血病蛋白质(Myeloid-Lymphoid Leukemia Protein);应激, 生理学(Stress, Physiological);串联重复序列(Tandem Repeat Sequences)
DOI
10.1182/blood-2012-02-412379
PMID
22740449
发布时间
2021-10-21
- 浏览29
Blood
1118-29页
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