换一批
换一批Notch-3 receptor activation drives inflammation and fibrosis following tubulointerstitial kidney injury.
第一作者：
Sonja,Djudjaj
第一单位：
Department of Nephrology and Hypertension, Diabetes and Endocrinology, Otto-von-Guericke University Magdeburg, Leipziger Strasse 44, 39120 Magdeburg, Germany.
作者：
主题词
动物(Animals);活组织检查(Biopsy);细胞系(Cell Line);细胞增殖(Cell Proliferation);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);纤维化(Fibrosis);人类(Humans);炎症(Inflammation);胞间信号肽类和蛋白质类(Intercellular Signaling Peptides and Proteins);肾(Kidney);膜蛋白质类(Membrane Proteins);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);肾炎, 间质性(Nephritis, Interstitial);大鼠(Rats);受体, Notch1(Receptor, Notch1);受体, Notch(Receptors, Notch);信号传导(Signal Transduction);转化生长因子β(Transforming Growth Factor beta);增量调节(Up-Regulation);输尿管梗阻(Ureteral Obstruction)
DOI
10.1002/path.4076
PMID
22806125
发布时间
2022-04-10
- 浏览12
The Journal of pathology
286-99页
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