Voltage-dependent anion channel-2 interaction with nitric oxide synthase enhances pulmonary artery endothelial cell nitric oxide production.
第一作者:
Cristina M,Alvira
第一单位:
Center of Excellence in Pulmonary Biology, Divisions of Pediatric Pulmonary, Asthma and Critical Care Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA.
作者:
主题词
动物(Animals);卡西霉素(Calcimycin);细胞, 培养的(Cells, Cultured);内皮细胞(Endothelial Cells);内皮, 血管(Endothelium, Vascular);基因表达(Gene Expression);组胺(Histamine);人类(Humans);婴儿, 新生(Infant, Newborn);一氧化氮合酶Ⅲ型(Nitric Oxide Synthase Type III);持续性胎儿循环综合征(Persistent Fetal Circulation Syndrome);蛋白质结合(Protein Binding);蛋白质相互作用图谱(Protein Interaction Mapping);肺动脉(Pulmonary Artery);绵羊(Sheep);电压依赖性阴离子通道1(Voltage-Dependent Anion Channel 1);电压依赖性阴离子通道2(Voltage-Dependent Anion Channel 2)
DOI
10.1165/rcmb.2011-0436OC
PMID
22842492
发布时间
2021-10-21
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