TRAF6-dependent Act1 phosphorylation by the IκB kinase-related kinases suppresses interleukin-17-induced NF-κB activation.

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作者： Fangfang,Qu ^[1] ; Hanchao,Gao ; Shu,Zhu ; Peiqing,Shi ; Yifan,Zhang ; Yan,Liu ; Bahija,Jallal ; Yihong,Yao ; Yufang,Shi ; Youcun,Qian

作者单位： The Key Laboratory of Stem Cell Biology, Institute of Health Sciences, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences & Shanghai Jiao Tong University School of Medicine, Shanghai, China. ^[1]

主题词动物(Animals);细胞系(Cell Line);连接蛋白43(Connexin 43);人类(Humans);I-κB激酶(I-kappa B Kinase);白细胞介素17(Interleukin-17);小鼠(Mice);肽碎片(Peptide Fragments);磷酰化(Phosphorylation);受体, 白细胞介素17(Receptors, Interleukin-17);TNF受体相关因子6(TNF Receptor-Associated Factor 6)

DOI 10.1128/MCB.00268-12

PMID 22851696

发布时间 2021-12-03

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Molecular and cellular biology

2012年32卷19期

3925-37页

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TRAF6-dependent Act1 phosphorylation by the IκB kinase-related kinases suppresses interleukin-17-induced NF-κB activation.

Molecular and cellular biology

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Molecular and cellular biology

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TRAF6-dependent Act1 phosphorylation by the IκB kinase-related kinases suppresses interleukin-17-induced NF-κB activation.

Molecular and cellular biology

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Molecular and cellular biology

相关期刊

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