Humanized mouse model of skin inflammation is characterized by disturbed keratinocyte differentiation and influx of IL-17A producing T cells.
第一作者:
Vivian L,de Oliveira
第一单位:
Laboratory of Medical Immunology, Department of Laboratory Medicine, Radboud University Nijmegen Medical Centre, Nijmegen, The Netherlands.
作者:
主题词
动物(Animals);抗原, 分化, T淋巴细胞(Antigens, Differentiation, T-Lymphocyte);CD4阳性T淋巴细胞(CD4-Positive T-Lymphocytes);CD8阳性T淋巴细胞(CD8-Positive T-Lymphocytes);细胞分化(Cell Differentiation);环孢菌素(Cyclosporine);疾病模型, 动物(Disease Models, Animal);弹性蛋白酶抑制因子(Elafin);基因表达调控(Gene Expression Regulation);人类(Humans);炎症(Inflammation);注射, 腹腔内(Injections, Intraperitoneal);白细胞介素17(Interleukin-17);角蛋白细胞(Keratinocytes);角蛋白质类(Keratins);Ki-67抗原(Ki-67 Antigen);L选择素(L-Selectin);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, SCID(Mice, SCID);西罗莫司(Sirolimus);皮肤(Skin);皮肤移植(Skin Transplantation);移植, 异种(Transplantation, Heterologous);β防御素(beta-Defensins)
DOI
10.1371/journal.pone.0045509
PMID
23094018
发布时间
2021-10-21
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PloS one
e45509页
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