Deletion of CDKAL1 affects high-fat diet-induced fat accumulation and glucose-stimulated insulin secretion in mice, indicating relevance to diabetes.
第一作者:
Tadashi,Okamura
第一单位:
Division of Animal Model, Department of Infectious Diseases, Research Institute, National Center for Global Health and Medicine, Tokyo, Japan.
作者:
医学主题词
脂肪组织(Adipose Tissue);成年人(Adult);动物(Animals);人体质量指数(Body Mass Index);体重(Body Weight);糖尿病, 2型(Diabetes Mellitus, Type 2);女(雌)性(Female);基因敲除技术(Gene Knockout Techniques);基因座(Genetic Loci);葡萄糖(Glucose);内环境稳定(Homeostasis);人类(Humans);胰岛素(Insulin);胰岛素抗药性(Insulin Resistance);小鼠(Mice);中年人(Middle Aged);神经组织蛋白质类(Nerve Tissue Proteins);肥胖症(Obesity);胰腺(Pancreas);表型(Phenotype);tRNA甲基转移酶类(tRNA Methyltransferases)
DOI
10.1371/journal.pone.0049055
PMID
23173044
发布时间
2021-10-21
- 浏览4
PloS one
e49055页
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