Hyperphosphatemia during spontaneous tumor lysis syndrome culminate in severe hypophosphatemia at the time of blast crisis of Phneg CML to acute myelomoncytic leukemia.

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作者： Ophira,Salomon ^[1] ; Eli J,Holtzman ; Pazit,Beckerman ; Camila,Avivi ; Luba,Trakhtenbrot ; Abraham,Kneller ; Tali,Tohami ; Yeroham,Kleinbaum ; Sara,Apter ; Ninette,Amariglio ; Ehud,Grossman ; Ginette,Schiby

作者单位： The Amalia Biron Research Institute of Thrombosis and Hemostasis, Sheba Medical Center and Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel. ophiras@sheba.health.gov.il. ^[1]

DOI 10.1186/2162-3619-1-24

PMID 23211092

发布时间 2021-10-21

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Experimental hematology & oncology

2012年1卷1期

24页

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Hyperphosphatemia during spontaneous tumor lysis syndrome culminate in severe hypophosphatemia at the time of blast crisis of Phneg CML to acute myelomoncytic leukemia.

Experimental hematology & oncology

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Experimental hematology & oncology

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Hyperphosphatemia during spontaneous tumor lysis syndrome culminate in severe hypophosphatemia at the time of blast crisis of Phneg CML to acute myelomoncytic leukemia.

Experimental hematology & oncology

相似文献

Experimental hematology & oncology

相关期刊

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