Hypothermia protects the brain from transient global ischemia/reperfusion by attenuating endoplasmic reticulum response-induced apoptosis through CHOP.
第一作者:
Xiaojie,Liu
第一单位:
Department of Anesthesiology, Qingdao Municipal Hospital, Shandong, China.
作者:
主题词
动物(Animals);细胞凋亡(Apoptosis);脑(Brain);脑缺血(Brain Ischemia);内质网(Endoplasmic Reticulum);热休克蛋白质类(Heat-Shock Proteins);海马(Hippocampus);低温, 人工(Hypothermia, Induced);免疫组织化学(Immunohistochemistry);原位缺口末端标记(In Situ Nick-End Labeling);男(雄)性(Male);线粒体(Mitochondria);大鼠(Rats);大鼠, Wistar(Rats, Wistar);再灌注损伤(Reperfusion Injury);时间因素(Time Factors);转录因子CHOP(Transcription Factor CHOP)
DOI
10.1371/journal.pone.0053431
PMID
23301071
发布时间
2023-11-04
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