Inflammation-mediated genetic and epigenetic alterations drive cancer development in the neighboring epithelium upon stromal abrogation of TGF-β signaling.

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作者： B R,Achyut ^[1] ; David A,Bader ; Ana I,Robles ; Darawalee,Wangsa ; Curtis C,Harris ; Thomas,Ried ; Li,Yang

作者单位： Laboratory of Cancer Biology and Genetics, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA. ^[1]

主题词动物(Animals);细胞凋亡(Apoptosis);乳腺肿瘤(Breast Neoplasms);癌, 鳞状细胞(Carcinoma, Squamous Cell);细胞转化, 肿瘤(Cell Transformation, Neoplastic);周期素依赖激酶抑制蛋白质类(Cyclin-Dependent Kinase Inhibitor Proteins);减量调节(Down-Regulation);后成说, 遗传(Epigenesis, Genetic);上皮细胞(Epithelial Cells);食管肿瘤(Esophageal Neoplasms);女(雌)性(Female);成纤维细胞(Fibroblasts);人类(Humans);炎症(Inflammation);小鼠(Mice);受体, 转化生长因子β(Receptors, Transforming Growth Factor beta);信号传导(Signal Transduction);间质细胞(Stromal Cells);转化生长因子β(Transforming Growth Factor beta)

DOI 10.1371/journal.pgen.1003251

PMID 23408900

发布时间 2021-12-03

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PLoS genetics

2013年9卷2期

e1003251页

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Inflammation-mediated genetic and epigenetic alterations drive cancer development in the neighboring epithelium upon stromal abrogation of TGF-β signaling.

PLoS genetics

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PLoS genetics

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Inflammation-mediated genetic and epigenetic alterations drive cancer development in the neighboring epithelium upon stromal abrogation of TGF-β signaling.

PLoS genetics

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PLoS genetics

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