换一批Genetic and pharmacological modifications of thrombin formation in apolipoprotein e-deficient mice determine atherosclerosis severity and atherothrombosis onset in a neutrophil-dependent manner.
第一作者：
Julian I,Borissoff
第一单位：
Laboratory for Clinical Thrombosis and Hemostasis, Department of Internal Medicine, Cardiovascular Research Institute Maastricht, Maastricht University Medical Center, Maastricht, The Netherlands. j.ilcheff@maastrichtuniversity.nl
作者：
医学主题词
动物(Animals);载脂蛋白E类(Apolipoproteins E);动脉粥样硬化(Atherosclerosis);苯咪唑类(Benzimidazoles);血液凝固(Blood Coagulation);疾病模型, 动物(Disease Models, Animal);疾病恶化(Disease Progression);女(雌)性(Female);血细胞生成(Hematopoiesis);炎症(Inflammation);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);中性白细胞(Neutrophils);表型(Phenotype);斑块, 动脉粥样硬化(Plaque, Atherosclerotic);吡啶类(Pyridines);活性氧(Reactive Oxygen Species);凝血酶(Thrombin);血栓形成(Thrombosis)
DOI
10.1371/journal.pone.0055784
PMID
23409043
发布时间
2021-10-21
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