KCNJ2 mutation in short QT syndrome 3 results in atrial fibrillation and ventricular proarrhythmia.

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作者： Makarand,Deo ^[1] ; Yanfei,Ruan ; Sandeep V,Pandit ; Kushal,Shah ; Omer,Berenfeld ; Andrew,Blaufox ; Marina,Cerrone ; Sami F,Noujaim ; Marco,Denegri ; José,Jalife ; Silvia G,Priori

作者单位： Department of Engineering, Norfolk State University, Norfolk, VA 23504, USA. ^[1]

主题词氨基酸取代(Amino Acid Substitution);心律失常, 心性(Arrhythmias, Cardiac);心房颤动(Atrial Fibrillation);儿童(Child);计算机模拟(Computer Simulation);HEK293细胞(HEK293 Cells);心脏传导系统(Heart Conduction System);心脏缺损, 先天性(Heart Defects, Congenital);人类(Humans);男(雄)性(Male);肌蛋白质类(Muscle Proteins);突变, 误义(Mutation, Missense);心肌(Myocardium);钾通道, 内向整流(Potassium Channels, Inwardly Rectifying);蛋白质多聚化(Protein Multimerization)

DOI 10.1073/pnas.1218154110

PMID 23440193

发布时间 2021-10-21

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Proceedings of the National Academy of Sciences of the United States of America

2013年110卷11期

4291-6页

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KCNJ2 mutation in short QT syndrome 3 results in atrial fibrillation and ventricular proarrhythmia.

Proceedings of the National Academy of Sciences of the United States of America

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Proceedings of the National Academy of Sciences of the United States of America

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KCNJ2 mutation in short QT syndrome 3 results in atrial fibrillation and ventricular proarrhythmia.

Proceedings of the National Academy of Sciences of the United States of America

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Proceedings of the National Academy of Sciences of the United States of America

相关期刊

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