COMMD1 interacts with the COOH terminus of NKCC1 in Calu-3 airway epithelial cells to modulate NKCC1 ubiquitination.

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作者： Laura,Smith ^[1] ; Paul,Litman ; Carole M,Liedtke

作者单位： Willard Alan Bernbaum, Center for Cystic Fibrosis Research, Departments of Pediatrics at Rainbow Babies and Children Hospital and Physiology and Biophysics, Case Western Reserve University, Cleveland, Ohio, USA. ^[1]

关键词 Na-K-2Cl cotransport binding assays biotinylation membrane expression silencing RNA truncation yeast two hybrid

主题词衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);细胞系(Cell Line);克隆, 分子(Cloning, Molecular);铜(Copper);上皮细胞(Epithelial Cells);基因表达调控(Gene Expression Regulation);人类(Humans);离子转运(Ion Transport);重组蛋白质类(Recombinant Proteins);钠钾氯化物协同转运子(Sodium-Potassium-Chloride Symporters);双杂交系统技术(Two-Hybrid System Techniques)

DOI 10.1152/ajpcell.00394.2012

PMID 23515529

发布时间 2021-10-21

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American journal of physiology. Cell physiology

2013年305卷2期

C133-46页

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COMMD1 interacts with the COOH terminus of NKCC1 in Calu-3 airway epithelial cells to modulate NKCC1 ubiquitination.

American journal of physiology. Cell physiology

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American journal of physiology. Cell physiology

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COMMD1 interacts with the COOH terminus of NKCC1 in Calu-3 airway epithelial cells to modulate NKCC1 ubiquitination.

American journal of physiology. Cell physiology

相似文献

American journal of physiology. Cell physiology

相关期刊

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