Chronic knockdown of the nucleus of the solitary tract AT1 receptors increases blood inflammatory-endothelial progenitor cell ratio and exacerbates hypertension in the spontaneously hypertensive rat.

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第一作者： Zhiying,Shan

第一单位： Department of Physiology and Functional Genomics, University of Florida, Gainesville, FL 32610, USA.

作者： Zhiying,Shan ^[1] ; Jasenka,Zubcevic ; Peng,Shi ; Joo Y,Jun ; Ying,Dong ; Tatiane M,Murça ; Gwyneth J,Lamont ; Adolfo,Cuadra ; Wei,Yuan ; Yanfei,Qi ; Qiuhong,Li ; Julian F R,Paton ; Michael J,Katovich ; Colin,Sumners ; Mohan K,Raizada

作者单位： Department of Physiology and Functional Genomics, University of Florida, Gainesville, FL 32610, USA. ^[1]

关键词 AT1R NTS baroreflex endothelial progenitor cells hypertension inflammation

主题词动物(Animals);血压(Blood Pressure);细胞, 培养的(Cells, Cultured);疾病恶化(Disease Progression);内皮细胞(Endothelial Cells);基因表达调控(Gene Expression Regulation);基因敲低技术(Gene Knockdown Techniques);高血压(Hypertension);炎症(Inflammation);RNA, 信使(RNA, Messenger);大鼠(Rats);大鼠, 近交SHR(Rats, Inbred SHR);大鼠, 近交WKY(Rats, Inbred WKY);受体, 血管紧张素, 1型(Receptor, Angiotensin, Type 1);孤束核(Solitary Nucleus);干细胞(Stem Cells)

DOI 10.1161/HYPERTENSIONAHA.111.00156

PMID 23547238

发布时间 2022-01-29

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Hypertension (Dallas, Tex.

2013年61卷6期

1328-33页

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Chronic knockdown of the nucleus of the solitary tract AT1 receptors increases blood inflammatory-endothelial progenitor cell ratio and exacerbates hypertension in the spontaneously hypertensive rat.

Hypertension (Dallas, Tex.

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Hypertension (Dallas, Tex.

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Chronic knockdown of the nucleus of the solitary tract AT1 receptors increases blood inflammatory-endothelial progenitor cell ratio and exacerbates hypertension in the spontaneously hypertensive rat.

Hypertension (Dallas, Tex.

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Hypertension (Dallas, Tex.

相关期刊

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