换一批A C-terminal fragment BIGH3 protein with an RGDRGD motif inhibits corneal neovascularization in vitro and in vivo.
第一作者：
Hongyan,Ge
第一单位：
Key Laboratory of Harbin Medical University Eye Center, Eye Hospital, First Affiliated Hospital, Harbin Medical University, 23 Youzheng Street, Nangang District, Harbin 150001, PR China.
作者：
医学主题词
氨基酸特征结构(Amino Acid Motifs);血管生成抑制剂(Angiogenesis Inhibitors);动物(Animals);细胞凋亡(Apoptosis);血管(Blood Vessels);印迹法, 蛋白质(Blotting, Western);半胱氨酸天冬氨酸蛋白酶(Caspases);细胞黏附(Cell Adhesion);细胞运动(Cell Movement);细胞增殖(Cell Proliferation);角膜新生血管化(Corneal Neovascularization);疾病模型, 动物(Disease Models, Animal);细胞外基质蛋白质类(Extracellular Matrix Proteins);流式细胞术(Flow Cytometry);整合素αVβ3(Integrin alphaVbeta3);诱变, 定点(Mutagenesis, Site-Directed);寡肽类(Oligopeptides);肽碎片(Peptide Fragments);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷酰化(Phosphorylation);质粒(Plasmids);蛋白质折叠(Protein Folding);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);兔(Rabbits);转化生长因子β(Transforming Growth Factor beta);血管内皮生长因子A(Vascular Endothelial Growth Factor A)
DOI
10.1016/j.exer.2013.03.014
PMID
23562678
发布时间
2022-04-08
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