Regulation of lung injury and fibrosis by p53-mediated changes in urokinase and plasminogen activator inhibitor-1.
第一作者:
Yashodhar P,Bhandary
第一单位:
Texas Lung Injury Institute, Center for Biomedical Research, University of Texas Health Science Center at Tyler, Tyler, Texas 75708, USA.
作者:
医学主题词
动物(Animals);博来霉素(Bleomycin);印迹法, 蛋白质(Blotting, Western);上皮细胞(Epithelial Cells);女(雌)性(Female);免疫组织化学(Immunohistochemistry);肺损伤(Lung Injury);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);纤溶酶原激活物抑制物1(Plasminogen Activator Inhibitor 1);肺泡(Pulmonary Alveoli);肺纤维化(Pulmonary Fibrosis);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53);尿激酶型纤溶酶原激活物(Urokinase-Type Plasminogen Activator)
DOI
10.1016/j.ajpath.2013.03.022
PMID
23665346
发布时间
2021-10-21
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