Translocation of iron from lysosomes to mitochondria during ischemia predisposes to injury after reperfusion in rat hepatocytes.
作者:
关键词
Cell deathFree radicalsIRIronIschemiaKRHKrebs–Ringer–HepesLTRLysoTracker RedLysosomeMCUMPTMitochondriaOH(?)PIROSRhDexTMRMV-ATPasechloromethyldichlorofluoresceinchloromethyldihydrodichlorofluorescein diacetatecmDCFcmH(2)DCF-DAhydroxyl radicalischemia–reperfusionmitochondrial Ca(2+) uniportermitochondrial membrane potentialmitochondrial permeability transitionpropidium iodidereactive oxygen speciesrhodamine–dextrantetramethylrhodamine methyl estervacuolar proton-pumping ATPaseΔΨ
主题词
动物(Animals);细胞凋亡(Apoptosis);钙(Calcium);肝细胞(Hepatocytes);铁(Iron);铁螯合剂(Iron Chelating Agents);溶酶体(Lysosomes);膜电位, 线粒体(Membrane Potential, Mitochondrial);线粒体, 肝(Mitochondria, Liver);通透性(Permeability);大鼠(Rats);活性氧(Reactive Oxygen Species);再灌注损伤(Reperfusion Injury)
DOI
10.1016/j.freeradbiomed.2013.05.004
PMID
23665427
发布时间
2024-06-10
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