换一批Provocation of an autoimmune response to cardiac voltage-gated sodium channel NaV1.5 induces cardiac conduction defects in rats.
第一作者：
Sevil,Korkmaz
第一单位：
Department of Cardiac Surgery, University of Heidelberg, Heidelberg, Germany.
作者：
关键词
AVBECGH&EI(Na)I(to)LVMMPNa(V)1.5SCN5ATIMPatrioventricular blockautoantibodiesautoimmunitycardiac sodium channelcardiac transient outward potassium currentcardiac voltage-gated sodium channel α-subunitcardiac voltage-gated sodium channel α-subunit genecardiac voltage-gated sodium-channel currentconduction defectdP/dt(max)dP/dt(min)electrocardiographyhematoxylin and eosinleft ventricularmatrix metalloproteinasemaximal slope of the diastolic pressure decrementmaximal slope of the systolic pressure incrementtissue inhibitor of metalloproteinase
主题词
氨基酸序列(Amino Acid Sequence);动物(Animals);心律失常, 心性(Arrhythmias, Cardiac);自身抗体(Autoantibodies);自身免疫疾病(Autoimmune Diseases);心脏传导系统(Heart Conduction System);男(雄)性(Male);分子序列数据(Molecular Sequence Data);随机分配(Random Allocation);大鼠(Rats);大鼠, 近交Lew(Rats, Inbred Lew)
DOI
10.1016/j.jacc.2013.04.041
PMID
23684688
发布时间
2013-07-22
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