换一批
换一批Increased proton leak and SOD2 expression in myotubes from obese non-diabetic subjects with a family history of type 2 diabetes.
作者：
关键词
2′,7′-dichlorfluorescein-diacetateANTCOXCSDCFH-DAFCCPFHGSHGSSGHGIIPAQIκKβJNKLGIMitochondrial proton leakMuscleOCROxidative phosphorylationOxidative stressPPARγRMRROSRQSOD2T2DMTMREUCP3adenine nucleotide translocasec-Jun N-terminal kinasecarbonyl cyanide 4-(trifluoromethoxy)phenylhydrazonecitrate synthasecytochrome C oxidasefamily historyhigh glucose, high insulin differentiation mediuminhibitor of NF-κB kinaseinternational physical activity questionnairelow glucose, low insulin differentiation mediummanganese superoxide dismutaseoxidized glutathioneoxygen consumption ratesperoxisome proliferator-activated receptor γreactive oxygen speciesreduced glutathionerespiratory quotientresting metabolic ratetetramethylrhodamine ethyl estertype 2 diabetes mellitusuncoupling protein 3
主题词
病例对照研究(Case-Control Studies);DNA, 线粒体(DNA, Mitochondrial);糖尿病, 2型(Diabetes Mellitus, Type 2);女(雌)性(Female);疾病遗传易感性(Genetic Predisposition to Disease);人类(Humans);胰岛素抗药性(Insulin Resistance);男(雄)性(Male);膜电位, 线粒体(Membrane Potential, Mitochondrial);中年人(Middle Aged);肌纤维, 骨骼(Muscle Fibers, Skeletal);肥胖症(Obesity);质子(Protons);超氧化物歧化酶(Superoxide Dismutase)
DOI
10.1016/j.bbadis.2013.05.008
PMID
23685312
发布时间
2016-11-26
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