Metabolic signatures of amyotrophic lateral sclerosis reveal insights into disease pathogenesis.

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作者： James C,Dodge ^[1] ; Christopher M,Treleaven ; Jonathan A,Fidler ; Thomas J,Tamsett ; Channa,Bao ; Michelle,Searles ; Tatyana V,Taksir ; Kuma,Misra ; Richard L,Sidman ; Seng H,Cheng ; Lamya S,Shihabuddin

作者单位： Genzyme, Framingham, MA 01701-9322, USA. jim.dodge@genzyme.com ^[1]

关键词 carbonic anhydrase electrolyte glucagon glycoproteins motor neuron disease

主题词酸中毒(Acidosis);肌萎缩侧索硬化(Amyotrophic Lateral Sclerosis);动物(Animals);疾病模型, 动物(Disease Models, Animal);疾病恶化(Disease Progression);糖原(Glycogen);人类(Humans);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);突变(Mutation);超氧化物歧化酶(Superoxide Dismutase)

DOI 10.1073/pnas.1308421110

PMID 23754387

发布时间 2021-10-21

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Proceedings of the National Academy of Sciences of the United States of America

2013年110卷26期

10812-7页

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Metabolic signatures of amyotrophic lateral sclerosis reveal insights into disease pathogenesis.

Proceedings of the National Academy of Sciences of the United States of America

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Proceedings of the National Academy of Sciences of the United States of America

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Metabolic signatures of amyotrophic lateral sclerosis reveal insights into disease pathogenesis.

Proceedings of the National Academy of Sciences of the United States of America

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Proceedings of the National Academy of Sciences of the United States of America

相关期刊

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