Mitochondrial oxidative stress corrupts coronary collateral growth by activating adenosine monophosphate activated kinase-α signaling.

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作者： Yuh Fen,Pung ^[1] ; Wai Johnn,Sam ; Kelly,Stevanov ; Molly,Enrick ; Chwen-Lih,Chen ; Christopher,Kolz ; Prashanth,Thakker ; James P,Hardwick ; Yeong-Renn,Chen ; Jason R B,Dyck ; Liya,Yin ; William M,Chilian

作者单位： Department of Integrative Medical Sciences, Northeast Ohio Medical University, Rootstown, OH 44272, USA. ^[1]

关键词 collateral circulation coronary circulation mitochondria reactive oxygen species

主题词 AMP活化蛋白激酶类(AMP-Activated Protein Kinases);动物(Animals);体重(Body Weight);细胞, 培养的(Cells, Cultured);冠状血管(Coronary Vessels);疾病模型, 动物(Disease Models, Animal);内皮细胞(Endothelial Cells);人类(Humans);缺血(Ischemia);线粒体(Mitochondria);心肌(Myocardium);氧化性应激(Oxidative Stress);大鼠(Rats);大鼠, 近交WKY(Rats, Inbred WKY);鱼藤酮(Rotenone);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);解偶联药(Uncoupling Agents)

DOI 10.1161/ATVBAHA.113.301591

PMID 23788766

发布时间 2021-12-03

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Arteriosclerosis, thrombosis, and vascular biology

2013年33卷8期

1911-9页

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Mitochondrial oxidative stress corrupts coronary collateral growth by activating adenosine monophosphate activated kinase-α signaling.

Arteriosclerosis, thrombosis, and vascular biology

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Arteriosclerosis, thrombosis, and vascular biology

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Mitochondrial oxidative stress corrupts coronary collateral growth by activating adenosine monophosphate activated kinase-α signaling.

Arteriosclerosis, thrombosis, and vascular biology

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Arteriosclerosis, thrombosis, and vascular biology

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