换一批
换一批Curcumin attenuates arsenic-induced hepatic injuries and oxidative stress in experimental mice through activation of Nrf2 pathway, promotion of arsenic methylation and urinary excretion.
作者：
关键词
ALTAREASTArsenicCurcuminDMAGSHGSH-PxHO-1LiverMDAMMAMethylatNADP(H) quinine oxidoreductase 1NQO1Nrf2Oxidative stressPMRSMRT-Asalanine amino transferaseantioxidant response elementaspartate aminotransferasedimethylarsinic acidglutathioneglutathione peroxidaseheme oxygenase-1iAsinorganic arsenicmalonaldehydemonomethylarsonic acidnuclear transcription factor NF-E2-related factor 2primary methylation ratiosecondary methylation ratiototal arsenic
主题词
动物(Animals);抗氧化剂(Antioxidants);砷(Arsenic);砷中毒(Arsenic Poisoning);姜黄素(Curcumin);营养保健品(Dietary Supplements);女(雌)性(Female);谷胱甘肽(Glutathione);血红素加氧酶-1(Heme Oxygenase-1);肝功能不全(Hepatic Insufficiency);肝(Liver);膜蛋白质类(Membrane Proteins);代谢解毒, Ⅱ期(Metabolic Detoxication, Phase II);甲基化(Methylation);小鼠(Mice);小鼠, 近交系(Mice, Inbred Strains);NAD(P)H脱氢酶(醌)(NAD(P)H Dehydrogenase (Quinone));NF-E2相关因子2(NF-E2-Related Factor 2);氧化还原(Oxidation-Reduction);氧化性应激(Oxidative Stress);随机分配(Random Allocation);信号传导(Signal Transduction);增量调节(Up-Regulation)
DOI
10.1016/j.fct.2013.07.032
PMID
23871787
发布时间
2022-03-31
- 浏览111
Food and chemical toxicology
739-47页
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