换一批
换一批Activation of STAT3 is involved in neuroprotection by electroacupuncture pretreatment via cannabinoid CB1 receptors in rats.
作者：
关键词
2,3,5-triphenyltetrazolium chloride2-AG2-arachidonylglycerolAEABehavioralEAERK1/2Ischemic toleranceMCAOMiddle cerebral artery occlusionN-arach-idonoylethanolamine-anandamideNeuroprotectionPKCεSTAT3Signal transducers and activators of transcription 3StrokeTTCTUNELdeoxyuridine triphosphate nick-end labeling.electroacupunctureextracellular signal-regulated kinases1/2middle cerebral artery occlusionprotein kinase C epsilon
主题词
动物(Animals);细胞凋亡(Apoptosis);半胱氨酸天冬氨酸蛋白酶3(Caspase 3);疾病模型, 动物(Disease Models, Animal);电针(Electroacupuncture);功能偏侧化(Functional Laterality);原位缺口末端标记(In Situ Nick-End Labeling);梗死, 大脑中动脉(Infarction, Middle Cerebral Artery);男(雄)性(Male);神经保护药(Neuroprotective Agents);肽类(Peptides);磷酸丙酮酸水合酶(Phosphopyruvate Hydratase);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, 大麻酚, CB1(Receptor, Cannabinoid, CB1);STAT3转录因子(STAT3 Transcription Factor);丝氨酸(Serine);增量调节(Up-Regulation)
DOI
10.1016/j.brainres.2013.07.006
PMID
23880371
发布时间
2013-08-19
- 浏览122
Brain research
154-64页
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