换一批Hypoxia-triggered m-calpain activation evokes endoplasmic reticulum stress and neuropathogenesis in a transgenic mouse model of Alzheimer's disease.
第一作者：
Chun-Yan,Wang
第一单位：
Department of Pathophysiology, Key Laboratory of Medical Cell Biology of Ministry of Education of China, China Medical University, Shenyang, China; Medical Research Laboratory, Jilin Medical College, Jilin, China.
作者：
医学主题词
阿尔茨海默病(Alzheimer Disease);淀粉样β蛋白前体(Amyloid beta-Protein Precursor);动物(Animals);卡配因(Calpain);细胞系, 肿瘤(Cell Line, Tumor);疾病模型, 动物(Disease Models, Animal);内质网(Endoplasmic Reticulum);女(雌)性(Female);海马(Hippocampus);人类(Humans);迷宫学习(Maze Learning);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);早老素1(Presenilin-1);随机分配(Random Allocation)
DOI
10.1111/cns.12151
PMID
23889979
发布时间
2021-10-21
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