Neuroprotective effects of edaravone on cognitive deficit, oxidative stress and tau hyperphosphorylation induced by intracerebroventricular streptozotocin in rats.

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作者： Shanshan,Zhou ^[1] ; Guichun,Yu ; Lijun,Chi ; Jiwei,Zhu ; Wei,Zhang ; Yan,Zhang ; Liming,Zhang

作者单位： Department of Neurology, The First Affiliated Hospital of Harbin Medical University, Harbin 150001, China. Electronic address: zhouss613008@126.com. ^[1]

关键词 Edaravone Oxidative stress Streptozotocin Tau hyperphosphorylation

主题词阿尔茨海默病(Alzheimer Disease);动物(Animals);安替比林(Antipyrine);认知障碍(Cognition Disorders);自由基清除剂(Free Radical Scavengers);输注, 脑室内(Infusions, Intraventricular);男(雄)性(Male);神经变性(Nerve Degeneration);神经保护药(Neuroprotective Agents);氧化性应激(Oxidative Stress);磷酰化(Phosphorylation);大鼠(Rats);链脲菌素(Streptozocin);tau蛋白质类(tau Proteins)

DOI 10.1016/j.neuro.2013.07.007

PMID 23932983

发布时间 2022-03-18

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Neurotoxicology

2013年38卷

136-45页

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Neuroprotective effects of edaravone on cognitive deficit, oxidative stress and tau hyperphosphorylation induced by intracerebroventricular streptozotocin in rats.

Neurotoxicology

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Neurotoxicology

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Neuroprotective effects of edaravone on cognitive deficit, oxidative stress and tau hyperphosphorylation induced by intracerebroventricular streptozotocin in rats.

Neurotoxicology

相似文献

Neurotoxicology

相关期刊

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