CCL5-glutamate interaction in central nervous system: Early and acute presynaptic defects in EAE mice.
作者:
关键词
4′-6′-diaminidino-2-phenylindoleACCCL5CD45CNSCSFCortexDAPIEAEEAE miceExperimental Autoimmune EncephalomyelitisGlutamate releaseHEIP(3)LFBLuxol Fast BlueMOGMSPLCRegulated upon activation normal T cells expressed and secreted, Tris-buffered salineSpinal cordTrisTris-(hydroxymethyl)-amino methane[(3)H]D-ASP[(3)H]d-aspartateadenylyl cyclasecAMPcentral nervous systemscerebrospinal fluidcluster of differentiation 45cyclic adenosine monophosphated.p.i.days post immunizationf.c.final concentrationhaematoxylin eosininositol-1,4,5-trisphosphatemultiple sclerosismyelin oligodendrocyte glycoproteinn.s.not significantphospholipase C
主题词
年龄因素(Age Factors);动物(Animals);动物, 新生(Animals, Newborn);中枢神经系统(Central Nervous System);趋化因子CCL5(Chemokine CCL5);D-天冬氨酸(D-Aspartic Acid);疾病模型, 动物(Disease Models, Animal);脑脊髓炎, 自身免疫性, 实验性(Encephalomyelitis, Autoimmune, Experimental);胞吐作用(Exocytosis);女(雌)性(Female);谷氨酸(Glutamic Acid);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);肽碎片(Peptide Fragments);氯化钾(Potassium Chloride);第二信使系统(Second Messenger Systems);突触体(Synaptosomes);时间因素(Time Factors);氚(Tritium)
DOI
10.1016/j.neuropharm.2013.07.037
PMID
23958452
发布时间
2014-11-20
- 浏览23
Neuropharmacology
337-46页
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