换一批
换一批Pleiotropic effects of the trichloroethylene-associated P81S VHL mutation on metabolism, apoptosis, and ATM-mediated DNA damage response.
第一作者：
Michelle C,Desimone
第一单位：
Affiliations of authors: Department of Genetics, North Carolina State University, Raleigh, NC (MCD, DWT); Department of Genetics (WKR) and Lineberger Cancer Center, University of North Carolina, Chapel Hill, NC (WKR, DWT).
作者：
主题词
动物(Animals);细胞凋亡(Apoptosis);精氨酸(Arginine);癌, 肾细胞(Carcinoma, Renal Cell);细胞周期蛋白质类(Cell Cycle Proteins);DNA损伤(DNA Damage);DNA结合蛋白质类(DNA-Binding Proteins);胚胎干细胞(Embryonic Stem Cells);谷氨酰胺(Glutamine);人类(Humans);缺氧诱导因子1(Hypoxia-Inducible Factor 1);免疫组织化学(Immunohistochemistry);肾肿瘤(Kidney Neoplasms);小鼠(Mice);职业暴露(Occupational Exposure);点突变(Point Mutation);脯氨酸(Proline);蛋白质阵列分析(Protein Array Analysis);丝氨酸(Serine);畸胎瘤(Teratoma);三氯乙烯(Trichloroethylene);肿瘤抑制蛋白质类(Tumor Suppressor Proteins);泛素蛋白连接酶类(Ubiquitin-Protein Ligases);脑视网膜血管瘤抑制蛋白质(Von Hippel-Lindau Tumor Suppressor Protein)
DOI
10.1093/jnci/djt226
PMID
23990666
发布时间
2022-04-30
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