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Plasmodium falciparum ATG8 implicated in both autophagy and apicoplast formation.

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第一作者： Andrew M,Tomlins

第一单位： Institute of Infection, Immunity and Inflammation; College of Medical, Veterinary and Life Sciences; University of Glasgow; Glasgow, UK.

作者： Andrew M,Tomlins ^[1] ; Fathia,Ben-Rached ^[2] ; Roderick Am,Williams ^[3] ; William R,Proto ^[4] ; Isabelle,Coppens ^[5] ; Ulrike,Ruch ^[6] ; Tim W,Gilberger ^[7] ; Graham H,Coombs ^[3] ; Jeremy C,Mottram ^[4] ; Sylke,Müller ^[1] ; Gordon,Langsley ^[2]

作者单位： Institute of Infection, Immunity and Inflammation; College of Medical, Veterinary and Life Sciences; University of Glasgow; Glasgow, UK. ^[1] INSERM U1016 CNRS UMR 8104; Institut Cochin; Université Paris Descartes; Sorbonne Paris Cité; Paris, France. ^[2] Strathclyde Institute of Pharmacy and Biomedical Sciences; University of Strathclyde; Glasgow, UK. ^[3] Wellcome Trust Centre for Molecular Parasitology; Institute of Infection, Immunity and Inflammation; College of Medical, Veterinary and Life Sciences; University of Glasgow; Glasgow, UK. ^[4] Department of Molecular Microbiology and Immunology; Johns Hopkins University Bloomberg School of Public Health; Baltimore, MD USA. ^[5] Department of Molecular Parasitology; Bernhard Nocht Institute for Tropical Medicine; Hamburg, Germany. ^[6] Department of Molecular Parasitology; Bernhard Nocht Institute for Tropical Medicine; Hamburg, Germany; M.G. DeGroote Institute for Infectious Disease Research and Department of Pathology and Molecular Medicine; McMaster University; Hamilton, Canada. ^[7]

关键词 ATG8 Plasmodium RAB7 apicoplast autophagy endosome food vacuole malaria