Smad3 inactivation and MiR-29b upregulation mediate the effect of carvedilol on attenuating the acute myocardium infarction-induced myocardial fibrosis in rat.
第一作者:
Jie-Ning,Zhu
第一单位:
Medical Research Department of Guangdong General Hospital, Guangdong Provincial Cardiovascular Institute, Guangdong Academy of Medical Sciences, Guangzhou, China.
作者:
主题词
动物(Animals);卡唑类(Carbazoles);强心药(Cardiotonic Agents);减量调节(Down-Regulation);成纤维细胞(Fibroblasts);纤维化(Fibrosis);心脏(Heart);男(雄)性(Male);微RNAs(MicroRNAs);心肌梗死(Myocardial Infarction);心肌(Myocardium);丙醇胺类(Propanolamines);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);Smad3蛋白质(Smad3 Protein);增量调节(Up-Regulation);心室功能, 左(Ventricular Function, Left)
DOI
10.1371/journal.pone.0075557
PMID
24086569
发布时间
2022-03-16
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