Fructose-1,6-diphosphate protects against epileptogenesis by modifying cation-chloride co-transporters in a model of amygdaloid-kindling temporal epilepticus.

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作者： Yao,Ding ^[1] ; Shan,Wang ; Yan,Jiang ; Yi,Yang ; Manman,Zhang ; Yi,Guo ; Shuang,Wang ; Mei-ping,Ding

作者单位： Department of Neurology, Second Affiliated Hospital, School of Medicine, Zhejiang University, No. 88, Jiefang Road, Hangzhou 310009, China. ^[1]

关键词 Amygdaloid kindling Antiepileptogenesis Cation-chloride cotransporters Fructose-1,6-diphosphate γ-Aminobutyric acid

主题词杏仁核(Amygdala);动物(Animals);抗惊厥药(Anticonvulsants);疾病模型, 动物(Disease Models, Animal);癫痫(Epilepsy);果糖-二磷酸酶(Fructose-Bisphosphatase);海马(Hippocampus);点燃效应, 神经病学(Kindling, Neurologic);男(雄)性(Male);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);钠钾氯化物协同转运子(Sodium-Potassium-Chloride Symporters);协同转运子(Symporters)

DOI 10.1016/j.brainres.2013.09.042

PMID 24095797

发布时间 2023-12-13

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Brain research

2013年1539卷

87-94页

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Fructose-1,6-diphosphate protects against epileptogenesis by modifying cation-chloride co-transporters in a model of amygdaloid-kindling temporal epilepticus.

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Fructose-1,6-diphosphate protects against epileptogenesis by modifying cation-chloride co-transporters in a model of amygdaloid-kindling temporal epilepticus.

Brain research

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Brain research

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