Autocrine VEGF signaling promotes proliferation of neoplastic Barrett's epithelial cells through a PLC-dependent pathway.-论文-万方医学网

第一作者： Qiuyang,Zhang

第一单位： Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Internal Medicine, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas.

作者： Qiuyang,Zhang ^[1] ; Chunhua,Yu ^[1] ; Sui,Peng ^[2] ; Hao,Xu ^[3] ; Ellen,Wright ^[4] ; Xi,Zhang ^[1] ; Xiaofang,Huo ^[1] ; Edaire,Cheng ^[5] ; Thai H,Pham ^[6] ; Kiyotaka,Asanuma ^[1] ; Kimmo J,Hatanpaa ^[7] ; Davood,Rezai ^[8] ; David H,Wang ^[9] ; Venetia,Sarode ^[7] ; Shelby,Melton ^[10] ; Robert M,Genta ^[11] ; Stuart J,Spechler ^[9] ; Rhonda F,Souza ^[12]

作者单位： Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Internal Medicine, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas. ^[1] Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Internal Medicine, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Division of Gastroenterology and Hepatology, The First Affiliated Hospital of Sun Yat-Sen University, Guangzhou, China. ^[2] Department of Internal Medicine, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas. ^[3] Department of Research and Development, VA North Texas Heath Care System, Dallas, Texas. ^[4] Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Internal Medicine, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Pediatrics, Children's Medical Center, Dallas, Texas. ^[5] Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Surgery, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas. ^[6] Department of Pathology, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas. ^[7] Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Research and Development, VA North Texas Heath Care System, Dallas, Texas. ^[8] Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Internal Medicine, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Harold C. Simmons Comprehensive Cancer Center, University of Texas Southwestern Medical Center, Dallas, Texas. ^[9] Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Pathology, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas. ^[10] Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Pathology, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Miraca Life Sciences, Inc., Irving, Texas. ^[11] Center for Esophageal Diseases, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Department of Internal Medicine, VA North Texas Health Care System and the University of Texas Southwestern Medical School, Dallas, Texas; Harold C. Simmons Comprehensive Cancer Center, University of Texas Southwestern Medical Center, Dallas, Texas. Electronic address: rhonda.souza@utsouthwestern.edu. ^[12]

主题词腺癌(Adenocarcinoma);动物(Animals);抗肿瘤药(Antineoplastic Agents);自身分泌性细胞间通讯(Autocrine Communication);Barrett食管(Barrett Esophagus);细胞系(Cell Line);细胞增殖(Cell Proliferation);食管肿瘤(Esophageal Neoplasms);女(雌)性(Female);人类(Humans);免疫组织化学(Immunohistochemistry);吲哚类(Indoles);MAP激酶信号系统(MAP Kinase Signaling System);小鼠(Mice);磷脂酶Cγ(Phospholipase C gamma);磷酰化(Phosphorylation);癌前状态(Precancerous Conditions);蛋白激酶C(Protein Kinase C);吡咯类(Pyrroles);治疗结果(Treatment Outcome);血管内皮生长因子A(Vascular Endothelial Growth Factor A);血管内皮生长因子受体2(Vascular Endothelial Growth Factor Receptor-2)

DOI 10.1053/j.gastro.2013.10.011

PMID 24120473

发布时间 2024-06-10

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Autocrine VEGF signaling promotes proliferation of neoplastic Barrett's epithelial cells through a PLC-dependent pathway.

Gastroenterology

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Autocrine VEGF signaling promotes proliferation of neoplastic Barrett's epithelial cells through a PLC-dependent pathway.

Gastroenterology

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Gastroenterology

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