Variation in chromatin accessibility in human kidney cancer links H3K36 methyltransferase loss with widespread RNA processing defects.
第一作者:
Jeremy M,Simon
第一单位:
Department of Genetics, University of North Carolina, Chapel Hill, North Carolina 27514, USA;
作者:
主题词
癌, 肾细胞(Carcinoma, Renal Cell);染色质(Chromatin);DNA结合蛋白质类(DNA-Binding Proteins);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);组蛋白赖氨酸N-甲基转移酶(Histone-Lysine N-Methyltransferase);人类(Humans);肾肿瘤(Kidney Neoplasms);突变(Mutation);核蛋白质类(Nuclear Proteins);RNA加工, 转录后(RNA Processing, Post-Transcriptional);RNA剪接(RNA Splicing);转录因子(Transcription Factors);肿瘤抑制蛋白质类(Tumor Suppressor Proteins);泛素硫酯酶(Ubiquitin Thiolesterase)
DOI
10.1101/gr.158253.113
PMID
24158655
发布时间
2021-10-21
- 浏览26
Genome research
241-50页
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