换一批Pulmonary M. tuberculosis infection delays Th1 immunity via immunoadaptor DAP12-regulated IRAK-M and IL-10 expression in antigen-presenting cells.
第一作者：
M,Jeyanathan
第一单位：
1] McMaster Immunology Research Centre and Department of Pathology and Molecular Medicine, Hamilton, Ontario, Canada [2] Michael G. DeGroote Institute for Infectious Disease Research, McMaster University, Hamilton, Ontario, Canada.
作者：
医学主题词
衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);动物(Animals);抗原呈递(Antigen Presentation);抗原呈递细胞(Antigen-Presenting Cells);细胞因子类(Cytokines);疾病模型, 动物(Disease Models, Animal);基因表达调控(Gene Expression Regulation);炎症介导素类(Inflammation Mediators);白细胞介素1受体相关激酶类(Interleukin-1 Receptor-Associated Kinases);白细胞介素10(Interleukin-10);肺(Lung);淋巴细胞活化(Lymphocyte Activation);男(雄)性(Male);小鼠(Mice);结核分枝杆菌(Mycobacterium tuberculosis);NF-κB(NF-kappa B);信号传导(Signal Transduction);Th1细胞(Th1 Cells);结核, 肺(Tuberculosis, Pulmonary)
DOI
10.1038/mi.2013.86
PMID
24172845
发布时间
2023-02-03
- 浏览8
Mucosal immunology
670-83页
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