换一批
换一批Myeloid-cell protein tyrosine phosphatase-1B deficiency in mice protects against high-fat diet and lipopolysaccharide-induced inflammation, hyperinsulinemia, and endotoxemia through an IL-10 STAT3-dependent mechanism.
第一作者：
Louise,Grant
第一单位：
Institute of Medical Sciences, University of Aberdeen, College of Life Sciences and Medicine, Foresterhill Health Campus, Aberdeen, United Kingdom.
作者：
主题词
脂肪组织, 白色(Adipose Tissue, White);动物(Animals);细胞系(Cell Line);膳食脂肪类(Dietary Fats);内毒素血症(Endotoxemia);基因表达调控, 酶学(Gene Expression Regulation, Enzymologic);葡萄糖(Glucose);内环境稳定(Homeostasis);高胰岛素血症(Hyperinsulinism);炎症(Inflammation);白细胞介素10(Interleukin-10);Janus激酶类(Janus Kinases);脂多糖类(Lipopolysaccharides);巨噬细胞(Macrophages);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);髓系细胞(Myeloid Cells);蛋白质酪氨酸磷酸酶, 非受体1型(Protein Tyrosine Phosphatase, Non-Receptor Type 1);STAT3转录因子(STAT3 Transcription Factor);信号传导(Signal Transduction);脾(Spleen)
DOI
10.2337/db13-0885
PMID
24186864
发布时间
2022-01-29
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Diabetes
456-70页
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