Antibodies trap tissue migrating helminth larvae and prevent tissue damage by driving IL-4Rα-independent alternative differentiation of macrophages.

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作者： Julia,Esser-von Bieren ^[1] ; Ilaria,Mosconi ; Romain,Guiet ; Alessandra,Piersgilli ; Beatrice,Volpe ; Fei,Chen ; William C,Gause ; Arne,Seitz ; J Sjef,Verbeek ; Nicola L,Harris

作者单位： Swiss Vaccine Research Institute and Global Health Institute, École Polytechnique Fédérale de Lausanne (EPFL), Lausanne, Switzerland. ^[1]

主题词动物(Animals);抗体, 蠕虫(Antibodies, Helminth);精氨酸酶(Arginase);细胞分化(Cell Differentiation);基因表达调控, 酶学(Gene Expression Regulation, Enzymologic);幼虫(Larva);巨噬细胞(Macrophages);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);疑似旋形线虫属(Nematospiroides dubius);受体, 细胞表面(Receptors, Cell Surface);信号传导(Signal Transduction);圆线目感染(Strongylida Infections)

DOI 10.1371/journal.ppat.1003771

PMID 24244174

发布时间 2022-03-23

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2013年9卷11期

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Antibodies trap tissue migrating helminth larvae and prevent tissue damage by driving IL-4Rα-independent alternative differentiation of macrophages.

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Antibodies trap tissue migrating helminth larvae and prevent tissue damage by driving IL-4Rα-independent alternative differentiation of macrophages.

PLoS pathogens

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PLoS pathogens

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