A phospho-BAD BH3 helix activates glucokinase by a mechanism distinct from that of allosteric activators.

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第一作者： Benjamin,Szlyk

第一单位： 1] Department of Cancer Biology, Dana-Farber Cancer Institute, Boston, Massachusetts, USA. [2].

作者： Benjamin,Szlyk ^[1] ; Craig R,Braun ^[2] ; Sanda,Ljubicic ^[3] ; Elaura,Patton ^[4] ; Gregory H,Bird ^[5] ; Mayowa A,Osundiji ^[4] ; Franz M,Matschinsky ^[6] ; Loren D,Walensky ^[7] ; Nika N,Danial ^[3]

作者单位： 1] Department of Cancer Biology, Dana-Farber Cancer Institute, Boston, Massachusetts, USA. [2]. ^[1] 1] Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, USA. [2]. ^[2] 1] Department of Cancer Biology, Dana-Farber Cancer Institute, Boston, Massachusetts, USA. [2] Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA. ^[3] Department of Cancer Biology, Dana-Farber Cancer Institute, Boston, Massachusetts, USA. ^[4] Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, USA. ^[5] Department of Biochemistry and Biophysics, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania, USA. ^[6] 1] Department of Pediatric Oncology, Dana-Farber Cancer Institute, Boston, Massachusetts, USA. [2] Department of Biological Chemistry and Molecular Pharmacology, Harvard Medical School, Boston, Massachusetts, USA. [3] Department of Pediatric Oncology, Children's Hospital, Boston, Massachusetts, USA. ^[7]

主题词别构调节(Allosteric Regulation);动物(Animals);酶激活(Enzyme Activation);葡糖激酶(Glucokinase);人类(Humans);磷蛋白类(Phosphoproteins)

DOI 10.1038/nsmb.2717

PMID 24317490

发布时间 2021-10-21

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准确一般字义偏差语意偏离译文作用小提交