换一批Calcium deficiency-induced and TRP channel-regulated IGF1R-PI3K-Akt signaling regulates abnormal epithelial cell proliferation.
第一作者：
W,Dai
第一单位：
Department of Molecular, Cellular and Developmental Biology, University of Michigan, Ann Arbor, MI 48109, USA.
作者：
医学主题词
动物(Animals);Caco-2细胞(Caco-2 Cells);钙(Calcium);细胞增殖(Cell Proliferation);上皮细胞(Epithelial Cells);人类(Humans);胰岛素样生长因子Ⅰ(Insulin-Like Growth Factor I);胰岛素样生长因子Ⅱ(Insulin-Like Growth Factor II);幼虫(Larva);寡核苷酸类, 反义(Oligonucleotides, Antisense);磷酸肌醇3-激酶(Phosphatidylinositol 3-Kinase);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);受体, IGF1型(Receptor, IGF Type 1);信号传导(Signal Transduction);钠钾交换ATP酶(Sodium-Potassium-Exchanging ATPase);TRPV阳离子通道(TRPV Cation Channels);卵黄囊(Yolk Sac);斑马鱼(Zebrafish);斑马鱼蛋白质类(Zebrafish Proteins)
DOI
10.1038/cdd.2013.177
PMID
24336047
发布时间
2022-04-08
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