Cardiac-specific Traf2 overexpression enhances cardiac hypertrophy through activating AKT/GSK3β signaling.
作者:
关键词
AKTAKT/glycogen synthase kinase 3βANPB-type natriuretic peptideBNPCardiomyocyteGAPDHGSK3βLVEDDLVESDPCRPressure overloadTACTraf2atrial natriuretic peptideconstriction of transverse aorticglyceraldehyde-3-phosphate dehydrogenaseleft ventricle end-diastolic diameterleft ventricle end-systolic diameterpolymerase chain reactiontumor necrosis factor receptor-associated factor 2β-MHCβ-myosin heavy chain
主题词
血管紧张素Ⅱ(Angiotensin II);动物(Animals);主动脉(Aorta);心脏扩大(Cardiomegaly);超声心动描记术(Echocardiography);糖原合成酶激酶3(Glycogen Synthase Kinase 3);心脏(Heart);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);肌细胞, 心脏(Myocytes, Cardiac);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);信号传导(Signal Transduction);TNF受体相关因子2(TNF Receptor-Associated Factor 2);增量调节(Up-Regulation);心室功能障碍, 左(Ventricular Dysfunction, Left)
DOI
10.1016/j.gene.2013.12.052
PMID
24378234
发布时间
2022-03-16
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Gene
225-31页
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