The phosphorylation of HIV-1 Gag by atypical protein kinase C facilitates viral infectivity by promoting Vpr incorporation into virions.
第一作者:
Ayumi,Kudoh
第一单位:
Department of Microbiology, Yokohama City University School of Medicine, Yokohama, Kanagawa, Japan. aryo@yokohama-cu.ac.jp.
作者:
主题词
HIV-1(HIV-1);宿主与病原体相互作用(Host-Pathogen Interactions);人类(Humans);磷酰化(Phosphorylation);蛋白激酶C(Protein Kinase C);蛋白质加工, 转译后(Protein Processing, Post-Translational);病毒粒子(Virion);病毒装配(Virus Assembly);gag基因产物, 人免疫缺陷病毒(gag Gene Products, Human Immunodeficiency Virus);vpr基因产物, 人免疫缺陷病毒(vpr Gene Products, Human Immunodeficiency Virus)
DOI
10.1186/1742-4690-11-9
PMID
24447338
发布时间
2021-10-21
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