Hepatic overexpression of steroid sulfatase ameliorates mouse models of obesity and type 2 diabetes through sex-specific mechanisms.

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作者： Mengxi,Jiang ^[1] ; Jinhan,He ; Heidi,Kucera ; Nilesh W,Gaikwad ; Bin,Zhang ; Meishu,Xu ; Robert M,O'Doherty ; Kyle W,Selcer ; Wen,Xie

作者单位： From the Center for Pharmacogenetics and Department of Pharmaceutical Sciences, University of Pittsburgh School of Pharmacy, Pittsburgh, Pennsylvania 15261. ^[1]

关键词 Diabetes Energy Metabolism Estrogen Obesity Steroid Hormone Transgenic Mice

主题词动物(Animals);糖尿病, 2型(Diabetes Mellitus, Type 2);能量代谢(Energy Metabolism);雌激素类(Estrogens);脂肪肝(Fatty Liver);女(雌)性(Female);胰岛素抗药性(Insulin Resistance);肝(Liver);男(雄)性(Male);小鼠(Mice);小鼠, 转基因(Mice, Transgenic);肥胖症(Obesity);甾醇硫酸酯酶(Steryl-Sulfatase);增量调节(Up-Regulation)

DOI 10.1074/jbc.M113.535914

PMID 24497646

发布时间 2022-03-10

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The Journal of biological chemistry

2014年289卷12期

8086-97页

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Hepatic overexpression of steroid sulfatase ameliorates mouse models of obesity and type 2 diabetes through sex-specific mechanisms.

The Journal of biological chemistry

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The Journal of biological chemistry

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Hepatic overexpression of steroid sulfatase ameliorates mouse models of obesity and type 2 diabetes through sex-specific mechanisms.

The Journal of biological chemistry

相似文献

The Journal of biological chemistry

相关期刊

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