换一批
换一批Alveolar epithelial cells in idiopathic pulmonary fibrosis display upregulation of TRAIL, DR4 and DR5 expression with simultaneous preferential over-expression of pro-apoptotic marker p53.
第一作者：
Khondoker M,Akram
第一单位：
Lung Research Group, Institute of Science and Technology in Medicine, Keele University UK.
作者：
主题词
细胞凋亡(Apoptosis);病例对照研究(Case-Control Studies);细胞增殖(Cell Proliferation);细胞周期蛋白D1(Cyclin D1);周期素依赖激酶抑制剂p21(Cyclin-Dependent Kinase Inhibitor p21);上皮细胞(Epithelial Cells);人类(Humans);特发性肺纤维化(Idiopathic Pulmonary Fibrosis);淋巴细胞(Lymphocytes);巨噬细胞, 肺泡(Macrophages, Alveolar);肌成纤维细胞(Myofibroblasts);肺泡(Pulmonary Alveoli);受体, TNF相关凋亡诱导配体(Receptors, TNF-Related Apoptosis-Inducing Ligand);受体, 肿瘤坏死因子(Receptors, Tumor Necrosis Factor);信号传导(Signal Transduction);细胞因子信号转导蛋白抑制因子(Suppressor of Cytokine Signaling Proteins);TNF相关凋亡诱导配体(TNF-Related Apoptosis-Inducing Ligand);肿瘤抑制蛋白质p53(Tumor Suppressor Protein p53);增量调节(Up-Regulation)
PMID
24551275
发布时间
2021-10-21
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