换一批
换一批BMI1 reprogrammes histone acetylation and enhances c-fos pathway via directly binding to Zmym3 in malignant myeloid progression.
第一作者：
Hongjie,Shen
第一单位：
Key Laboratory of Thrombosis and Hemostasis of Ministry of Health, Jiangsu Institute of Hematology, The First Affiliated Hospital of Soochow University, Soochow, China.
作者：
主题词
乙酰化作用(Acetylation);青少年(Adolescent);成年人(Adult);老年人(Aged);老年人, 80以上(Aged, 80 and over);细胞凋亡(Apoptosis);印迹法, 蛋白质(Blotting, Western);酪酸(Butyric Acid);病例对照研究(Case-Control Studies);细胞分化(Cell Differentiation);细胞增殖(Cell Proliferation);细胞, 培养的(Cells, Cultured);染色质免疫沉淀法(Chromatin Immunoprecipitation);女(雌)性(Female);流式细胞术(Flow Cytometry);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);组胺拮抗药(Histamine Antagonists);组蛋白类(Histones);人类(Humans);白血病, 髓系, 慢性, BCR-ABL阳性(Leukemia, Myelogenous, Chronic, BCR-ABL Positive);男(雄)性(Male);中年人(Middle Aged);骨髓增生异常综合征(Myelodysplastic Syndromes);核蛋白质类(Nuclear Proteins);蛋白质结合(Protein Binding);原癌基因蛋白质c-fos(Proto-Oncogene Proteins c-fos);RNA, 信使(RNA, Messenger);逆转录聚合酶链反应(Reverse Transcriptase Polymerase Chain Reaction);十四酰佛波乙酯(Tetradecanoylphorbol Acetate);青年人(Young Adult)
DOI
10.1111/jcmm.12246
PMID
24571310
发布时间
2021-10-21
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