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I(f) blocking potency of ivabradine is preserved under elevated endotoxin levels in human atrial myocytes.

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第一作者： Susanne,Scheruebel

第一单位： Institute of Biophysics, Medical University of Graz, Harrachgasse 21, A-8010 Graz, Austria.

作者： Susanne,Scheruebel ^[1] ; Chintan N,Koyani ^[2] ; Seth,Hallström ^[3] ; Petra,Lang ^[1] ; Dieter,Platzer ^[1] ; Heinrich,Mächler ^[4] ; Karl,Lohner ^[5] ; Ernst,Malle ^[2] ; Klaus,Zorn-Pauly ^[6] ; Brigitte,Pelzmann ^[7]

作者单位： Institute of Biophysics, Medical University of Graz, Harrachgasse 21, A-8010 Graz, Austria. ^[1] Institute of Molecular Biology and Biochemistry, Medical University of Graz, Harrachgasse 21, A-8010 Graz, Austria. ^[2] Institute of Physiological Chemistry, Medical University of Graz, Harrachgasse 21, A-8010 Graz, Austria. ^[3] Division of Cardiac Surgery, Medical University of Graz, Auenbruggerplatz, A-8010 Graz, Austria. ^[4] Institute of Molecular Biosciences, Biophysics Division, University of Graz, Schmiedlstrasse 6, A-8042 Graz, Austria. ^[5] Institute of Biophysics, Medical University of Graz, Harrachgasse 21, A-8010 Graz, Austria. Electronic address: klaus.zornpauly@medunigraz.at. ^[6] Institute of Biophysics, Medical University of Graz, Harrachgasse 21, A-8010 Graz, Austria. Electronic address: brigitte.pelzmann@medunigraz.at. ^[7]

关键词 HCN channel Human pacemaker current Ivabradine Lipopolysaccharide Patch clamp Sinoatrial cell model