Blockade of gap junction hemichannel protects secondary spinal cord injury from activated microglia-mediated glutamate exitoneurotoxicity.
第一作者:
Daisuke,Umebayashi
第一单位:
1 Department of Neurosurgery, Nagoya University School of Medicine , Nagoya, Japan .
作者:
主题词
动物(Animals);兴奋性氨基酸拮抗剂(Excitatory Amino Acid Antagonists);兴奋性氨基酸类(Excitatory Amino Acids);女(雌)性(Female);缝隙接合部(Gap Junctions);谷氨酸(Glutamic Acid);免疫组织化学(Immunohistochemistry);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小神经胶质细胞(Microglia);神经保护药(Neuroprotective Agents);逆转录聚合酶链反应(Reverse Transcriptase Polymerase Chain Reaction);脊髓损伤(Spinal Cord Injuries)
DOI
10.1089/neu.2013.3223
PMID
24588281
发布时间
2021-10-21
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Journal of neurotrauma
1967-74页
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