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RUNX family members are covalently modified and regulated by PIAS1-mediated sumoylation.

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第一作者： J-H,Kim

第一单位： Department of Biochemistry, College of Medicine, Chungbuk National University, Cheongju, South Korea.

作者： J-H,Kim ^[1] ; J-W,Jang ^[1] ; Y-S,Lee ^[1] ; J-W,Lee ^[1] ; X-Z,Chi ^[1] ; Y-H,Li ^[1] ; M-K,Kim ^[1] ; D-M,Kim ^[1] ; B-S,Choi ^[1] ; J,Kim ^[1] ; H-M,Kim ^[2] ; A,van Wijnen ^[3] ; Ily,Park ^[4] ; S-C,Bae ^[1]

作者单位： Department of Biochemistry, College of Medicine, Chungbuk National University, Cheongju, South Korea. ^[1] Gachon Institute of Pharmaceutical Sciences, Gachon University, Incheon, South Korea. ^[2] Department of Orthopedic Surgery and Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN, USA. ^[3] College of Pharmacy, Chungbuk National University, Cheongju, South Korea. ^[4]

DOI 10.1038/oncsis.2014.15

PMID 24777122

发布时间 2021-10-21

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Oncogenesis

2014年3卷

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RUNX family members are covalently modified and regulated by PIAS1-mediated sumoylation.

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RUNX family members are covalently modified and regulated by PIAS1-mediated sumoylation.

Oncogenesis

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Oncogenesis

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