KIF14 promotes AKT phosphorylation and contributes to chemoresistance in triple-negative breast cancer.
第一作者:
Stina M,Singel
第一单位:
Division of Hematology-Oncology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX, USA; Department of Cell Biology University of Texas Southwestern Medical Center, Dallas, TX, USA.
作者:
主题词
BRCA1蛋白质(BRCA1 Protein);细胞增殖(Cell Proliferation);抗药性, 肿瘤(Drug Resistance, Neoplasm);女(雌)性(Female);基因表达调控, 肿瘤(Gene Expression Regulation, Neoplastic);基因敲低技术(Gene Knockdown Techniques);杂合子(Heterozygote);人类(Humans);肿瘤辅助疗法(Neoadjuvant Therapy);癌基因蛋白质类(Oncogene Proteins);磷酰化(Phosphorylation);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt)
DOI
10.1016/j.neo.2014.03.008
PMID
24784001
发布时间
2022-04-08
- 浏览26

Neoplasia (New York, N.Y.)
247-56, 256.e2页
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