The noncoding RNA IPW regulates the imprinted DLK1-DIO3 locus in an induced pluripotent stem cell model of Prader-Willi syndrome.

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作者： Yonatan,Stelzer ^[1] ; Ido,Sagi ^[1] ; Ofra,Yanuka ^[1] ; Rachel,Eiges ^[2] ; Nissim,Benvenisty ^[1]

作者单位： Stem Cell Unit, Department of Genetics, Institute of Life Sciences, The Hebrew University, Jerusalem, Israel. ^[1] Stem Cell Research Laboratory, Shaare Zedek Medical Center, The Hebrew University, Jerusalem, Israel. ^[2]

主题词青少年(Adolescent);脑(Brain);钙结合蛋白质类(Calcium-Binding Proteins);染色质(Chromatin);染色体畸变(Chromosome Aberrations);DNA甲基化(DNA Methylation);减量调节(Down-Regulation);成纤维细胞(Fibroblasts);基因表达谱(Gene Expression Profiling);全基因组关联研究(Genome-Wide Association Study);基因组印迹(Genomic Imprinting);人类(Humans);诱导多能干细胞(Induced Pluripotent Stem Cells);婴儿(Infant);胞间信号肽类和蛋白质类(Intercellular Signaling Peptides and Proteins);碘化物过氧化物酶(Iodide Peroxidase);男(雄)性(Male);膜蛋白质类(Membrane Proteins);寡核苷酸序列分析(Oligonucleotide Array Sequence Analysis);表型(Phenotype);Prader-Willi综合征(Prader-Willi Syndrome);RNA, 未翻译(RNA, Untranslated);转录, 遗传(Transcription, Genetic)

DOI 10.1038/ng.2968

PMID 24816254

发布时间 2021-10-21

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Nature genetics

2014年46卷6期

551-7页

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The noncoding RNA IPW regulates the imprinted DLK1-DIO3 locus in an induced pluripotent stem cell model of Prader-Willi syndrome.

Nature genetics

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The noncoding RNA IPW regulates the imprinted DLK1-DIO3 locus in an induced pluripotent stem cell model of Prader-Willi syndrome.

Nature genetics

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Nature genetics

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