HDL inhibits the effects of oxidized phospholipids on endothelial cell gene expression via multiple mechanisms.

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作者： Benjamin,Emert ^[1] ; Yehudit,Hasin-Brumshtein ^[1] ; James R,Springstead ^[2] ; Ladan,Vakili ^[1] ; Judith A,Berliner ^[3] ; Aldons J,Lusis ^[4]

作者单位： Department of Medicine, Division of Cardiology University of California, Los Angeles, Los Angeles, CA 90095. ^[1] Department of Chemical Engineering, Western Michigan University, Kalamazoo, MI 49008. ^[2] Department of Medicine, Division of Cardiology University of California, Los Angeles, Los Angeles, CA 90095 Departments of Pathology, University of California, Los Angeles, Los Angeles, CA 90095. ^[3] Department of Medicine, Division of Cardiology University of California, Los Angeles, Los Angeles, CA 90095 Departments of Pathology, University of California, Los Angeles, Los Angeles, CA 90095 Human Genetics University of California, Los Angeles, Los Angeles, CA 90095 Microbiology, Immunology and Molecular Genetics, University of California, Los Angeles, Los Angeles, CA 90095. ^[4]

关键词 high density lipoprotein inflammatory signaling minimally modified low density lipoprotein platelet-activating factor acetylhydrolase scavenger receptor class B type I

主题词细胞, 培养的(Cells, Cultured);内皮细胞(Endothelial Cells);基因表达调控(Gene Expression Regulation);人类(Humans);脂蛋白类, HDL(Lipoproteins, HDL);磷脂类(Phospholipids)

DOI 10.1194/jlr.M047738

PMID 24859737

发布时间 2021-10-21

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